Disease Info Card

Adult-onset Still Disease

Information about Adult-onset Still Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Adult-onset Still Disease

Most recent studies have shown that Adult-onset Still Disease shares some biological mechanisms with arthralgia, arthritis, arthritis-juvenile-rheumatoid, autoimmune-diseases, autoimmune-reaction, exanthema, fever-of-unknown-origin, hepatitis, inflammation, inflammatory-disorder, juvenile-onset-still-disease, leukocytosis, lupus-erythematosus-systemic, lymphohistiocytosis-hemophagocytic, lymphoma, pain, polyarthritis, rheumatism, rheumatoid-arthritis, sore-throat.

Among the many pathways, these few ones have gauged particular interests from scientists studying Adult-onset Still Disease, and have been seen in publications frequently: Acute Inflammatory Response, Aging, Cell Activation, Coagulation, Cytokine Production, Cytolysis, Diapedesis, Glycosylation, Hormone Secretion, Hypersensitivity, Immune Response, Inflammatory Response, Innate Immune Response, Macrophage Activation, Pathogenesis, Phagocytosis, Pigmentation, Response To Methotrexate, Secretion, T Cell Activation

Quite a number of genes have been found to play important roles in Adult-onset Still Disease, such as ADAMTS13, CALCA, CRP, CSRP1, ESR1, IL18, IL1B, IL1RN, IL2, IL4, IL6, LYZ, NOD2, SLC17A5, SLC25A10, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.